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Department of Cardiovascular Sciences,
Centre for Molecular and Vascular Biology,
Department of Cardiovascular Sciences, Centre for Molecular and Vascular Biology
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Correction of endothelial dysfunction after selective homocysteine lowering gene therapy reduces arterial thrombogenicity but has no effect on atherogenesis.
Journal of molecular medicine (Berlin, Germany) Oct, 2011 | Pubmed ID: 21688073
Protein tyrosine phosphatase SHP2 mediates chronic insulin-induced endothelial inflammation.
Arteriosclerosis, thrombosis, and vascular biology Aug, 2012 | Pubmed ID: 22628433
Lipid lowering and HDL raising gene transfer increase endothelial progenitor cells, enhance myocardial vascularity, and improve diastolic function.
PloS one , 2012 | Pubmed ID: 23056485
Selective homocysteine lowering gene transfer improves infarct healing, attenuates remodelling, and enhances diastolic function after myocardial infarction in mice.
PloS one , 2013 | Pubmed ID: 23675503
The liver as a target organ for gene therapy: state of the art, challenges, and future perspectives.
Pharmaceuticals (Basel, Switzerland) , 2012 | Pubmed ID: 24281341
The Impact of Lipoproteins on Wound Healing: Topical HDL Therapy Corrects Delayed Wound Healing in Apolipoprotein E Deficient Mice.
Pharmaceuticals (Basel, Switzerland) , 2014 | Pubmed ID: 24705596
Catholic University of Leuven
Ilayaraja Muthuramu1,
Marleen Lox1,
Frank Jacobs1,
Bart De Geest1
1Department of Cardiovascular Sciences, Centre for Molecular and Vascular Biology, Catholic University of Leuven
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