Juan T. Borda

[Renal injury in rats poisoned by venom of Bothrops neuwiedii diporus from Argentina].

Cell tropism of simian immunodeficiency virus in culture is not predictive of in vivo tropism or pathogenesis.

Visualizing cytokine-secreting cells in situ in the rhesus macaque model of chronic gut inflammation.

Characterization of cytolethal distending toxin of campylobacter species isolated from captive macaque monkeys.

Serial electrophysiologic studies in rhesus monkeys with Krabbe disease.

A decline in CCL3-5 chemokine gene expression during primary simian-human immunodeficiency virus infection.

Gastrointestinal disease in simian immunodeficiency virus-infected rhesus macaques is characterized by proinflammatory dysregulation of the interleukin-6-Janus kinase/signal transducer and activator of transcription3 pathway.

Clinical and immunopathologic alterations in rhesus macaques affected with globoid cell leukodystrophy.

CD163, a marker of perivascular macrophages, is up-regulated by microglia in simian immunodeficiency virus encephalitis after haptoglobin-hemoglobin complex stimulation and is suggestive of breakdown of the blood-brain barrier.

A non-human primate model for gluten sensitivity.

CCAAT/enhancer binding protein beta is a major mediator of inflammation and viral replication in the gastrointestinal tract of simian immunodeficiency virus-infected rhesus macaques.

Interaction of the Lyme disease spirochete Borrelia burgdorferi with brain parenchyma elicits inflammatory mediators from glial cells as well as glial and neuronal apoptosis.

The level of monocyte turnover predicts disease progression in the macaque model of AIDS.

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

Visualization of transepithelial passage of the immunogenic 33-residue peptide from alpha-2 gliadin in gluten-sensitive macaques.

Neurokinin-1 receptor (NK1-R) expression in the brains of SIV-infected rhesus macaques: implications for substance P in NK1-R immune cell trafficking into the CNS.

Persistence of Borrelia burgdorferi in Rhesus Macaques following Antibiotic Treatment of Disseminated Infection.