Marie-Christine Mariol

The MYST domain acetyltransferase Chameau functions in epigenetic mechanisms of transcriptional repression.

DWnt4 regulates cell movement and focal adhesion kinase during Drosophila ovarian morphogenesis.

Overexpression of dystrobrevin delays locomotion defects and muscle degeneration in a dystrophin-deficient Caenorhabditis elegans.

Genetic evidence for a dystrophin-glycoprotein complex (DGC) in Caenorhabditis elegans.

Characterization of K(+) currents using an in situ patch clamp technique in body wall muscle cells from Caenorhabditis elegans.

The L-type voltage-dependent Ca2+ channel EGL-19 controls body wall muscle function in Caenorhabditis elegans.

The stn-1 syntrophin gene of C.elegans is functionally related to dystrophin and dystrobrevin.

Patch clamp study of the UNC-105 degenerin and its interaction with the LET-2 collagen in Caenorhabditis elegans muscle.

The SLO-1 BK channel of Caenorhabditis elegans is critical for muscle function and is involved in dystrophin-dependent muscle dystrophy.

Blocking of striated muscle degeneration by serotonin in C. elegans.

Dystrophin-dependent muscle degeneration requires a fully functional contractile machinery to occur in C. elegans.

DYC-1, a protein functionally linked to dystrophin in Caenorhabditis elegans is associated with the dense body, where it interacts with the muscle LIM domain protein ZYX-1.

ZYX-1, the unique zyxin protein of Caenorhabditis elegans, is involved in dystrophin-dependent muscle degeneration.

Chemical genetics unveils a key role of mitochondrial dynamics, cytochrome c release and IP3R activity in muscular dystrophy.