Baskaran Thyagarajan

Nitric oxide inhibits capacitative Ca2+ entry by suppression of mitochondrial Ca2+ handling.

Intermittent claudication: an overview.

Rapidly inducible changes in phosphatidylinositol 4,5-bisphosphate levels influence multiple regulatory functions of the lipid in intact living cells.

Dual regulation of TRPV1 by phosphoinositides.

Hydrolysis of phosphatidylinositol 4,5-bisphosphate mediates calcium-induced inactivation of TRPV6 channels.

Phospholipase C mediated modulation of TRPV1 channels.

Phospholipase C-mediated regulation of transient receptor potential vanilloid 6 channels: implications in active intestinal Ca2+ transport.

Inorganic polyphosphate modulates TRPM8 channels.

Capsaicin protects mouse neuromuscular junctions from the neuroparalytic effects of botulinum neurotoxin a.

Effects of hydroxamate metalloendoprotease inhibitors on botulinum neurotoxin A poisoned mouse neuromuscular junctions.

Recombinant botulinum neurotoxin A heavy chain-based delivery vehicles for neuronal cell targeting.

Acetylcholinesterase deficiency contributes to neuromuscular junction dysfunction in type 1 diabetic neuropathy.

Effects of enzymatically inactive recombinant botulinum neurotoxin type A at the mouse neuromuscular junctions.

Cotrimoxazole and neonatal kernicterus: a review.

Acute and chronic effects of botulinum neurotoxin a on the mammalian neuromuscular junction.

Capsaicin modulates acetylcholine release at the myoneural junction.

The TRPM8 protein is a testosterone receptor: I. Biochemical evidence for direct TRPM8-testosterone interactions.

The TRPM8 protein is a testosterone receptor: II. Functional evidence for an ionotropic effect of testosterone on TRPM8.

Capsaicin induces browning of white adipose tissue and counters obesity by activating TRPV1 channel-dependent mechanisms.

Preparation and Evaluation of PLGA-Coated Capsaicin Magnetic Nanoparticles.

Perturbation to Cholesterol at the Neuromuscular Junction confers botulinum neurotoxin A sensitivity to neonatal mice.

Beiging of white adipose tissue as a therapeutic strategy for weight loss in humans.