Padmamalini Baskaran

Desensitization of soluble guanylyl cyclase, the NO receptor, by S-nitrosylation.

PAS-mediated dimerization of soluble guanylyl cyclase revealed by signal transduction histidine kinase domain crystal structure.

Structure of cinaciguat (BAY 58-2667) bound to Nostoc H-NOX domain reveals insights into heme-mimetic activation of the soluble guanylyl cyclase.

Aspartate 102 in the heme domain of soluble guanylyl cyclase has a key role in NO activation.

Inhibition of nitric oxide-stimulated vasorelaxation by carbon monoxide-releasing molecules.

Identification of residues in the heme domain of soluble guanylyl cyclase that are important for basal and stimulated catalytic activity.

Soluble guanylyl cyclase is a target of angiotensin II-induced nitrosative stress in a hypertensive rat model.

Protein disulfide-isomerase interacts with soluble guanylyl cyclase via a redox-based mechanism and modulates its activity.

Effects of enzymatically inactive recombinant botulinum neurotoxin type A at the mouse neuromuscular junctions.

Cyclophilin A is required for angiotensin II-induced p47phox translocation to caveolae in vascular smooth muscle cells.

Acute and chronic effects of botulinum neurotoxin a on the mammalian neuromuscular junction.

Acetylation of cyclophilin A is required for its secretion and vascular cell activation.

Cyclophilin A is an important mediator of platelet function by regulating integrin αIIbβ3 bidirectional signalling.

Capsaicin modulates acetylcholine release at the myoneural junction.

The TRPM8 protein is a testosterone receptor: I. Biochemical evidence for direct TRPM8-testosterone interactions.

The TRPM8 protein is a testosterone receptor: II. Functional evidence for an ionotropic effect of testosterone on TRPM8.

Capsaicin induces browning of white adipose tissue and counters obesity by activating TRPV1 channel-dependent mechanisms.

Preparation and Evaluation of PLGA-Coated Capsaicin Magnetic Nanoparticles.

Perturbation to Cholesterol at the Neuromuscular Junction confers botulinum neurotoxin A sensitivity to neonatal mice.