Sheng Wu

Rescue of obesity-induced infertility in female mice due to a pituitary-specific knockout of the insulin receptor.

Jak2 is necessary for neuroendocrine control of female reproduction.

Reproductive tissues maintain insulin sensitivity in diet-induced obesity.

Signaling of cytokines is important in regulation of GnRH neurons.

Estrogen receptor-β in the gonadotropin-releasing hormone neuron.

Both estrogen receptor α and β stimulate pituitary GH gene expression.

Obesity-induced infertility and hyperandrogenism are corrected by deletion of the insulin receptor in the ovarian theca cell.

The regulation of reproductive neuroendocrine function by insulin and insulin-like growth factor-1 (IGF-1).

Androgen Receptor in the Ovary Theca Cells Plays a Critical Role in Androgen-Induced Reproductive Dysfunction.

Low-Dose Dihydrotestosterone Drives Metabolic Dysfunction via Cytosolic and Nuclear Hepatic Androgen Receptor Mechanisms.

The impact of androgen actions in neurons on metabolic health and disease.

Female Offspring From Chronic Hyperandrogenemic Dams Exhibit Delayed Puberty and Impaired Ovarian Reserve.

Insulin signaling displayed a differential tissue-specific response to low-dose dihydrotestosterone in female mice.

Bitter Taste Receptor Ligand Improves Metabolic and Reproductive Functions in a Murine Model of PCOS.

VCAM1 Is Induced in Ovarian Theca and Stromal Cells in a Mouse Model of Androgen Excess.

Gonadotrope androgen receptor mediates pituitary responsiveness to hormones and androgen-induced subfertility.

Androgen-induced insulin resistance is ameliorated by deletion of hepatic androgen receptor in females.