Marcin Ufnal

Enhanced pressor response to centrally administered vasopressin in WKY rats on high sodium diet.

Blockade of angiotensin II AT1 receptors inhibits pressor action of centrally administered interleukin-1beta in Sprague Dawley rats.

Inhibition of brain nitric oxide synthesis enhances and prolongs the hypertensive effect of centrally administered interleukin-1beta in rats.

Centrally administered interleukin-1 beta sensitizes to the central pressor action of angiotensin II.

Differential sensitisation to central cardiovascular effects of angiotensin II in rats with a myocardial infarct: relevance to stress and interaction with vasopressin.

Exogenous hydrogen sulfide produces hemodynamic effects by triggering central neuroregulatory mechanisms.

Interleukin-1 receptor antagonist reduces the magnitude of the pressor response to acute stress.

Simvastatin reduces pressor response to centrally administered angiotensin II.

The role of brain gaseous transmitters in the regulation of the circulatory system.

Oral simvastatin reduces the hypertensive response to air-jet stress.

Essential hypertension--is erroneous receptor output to blame?

Early high-sodium solid diet does not affect sodium intake, sodium preference, blood volume and blood pressure in adult Wistar-Kyoto rats.

Blood borne hormones in a cross-talk between peripheral and brain mechanisms regulating blood pressure, the role of circumventricular organs.

Statins, the renin-angiotensin-aldosterone system and hypertension - a tale of another beneficial effect of statins.

Exogenous hydrogen sulfide causes different hemodynamic effects in normotensive and hypertensive rats via neurogenic mechanisms.

Trimethylamine-N-oxide: a carnitine-derived metabolite that prolongs the hypertensive effect of angiotensin II in rats.

TNF and angiotensin type 1 receptors interact in the brain control of blood pressure in heart failure.

The effect of simvastatin and pravastatin on arterial blood pressure, baroreflex, vasoconstrictor, and hypertensive effects of angiotensin II in Sprague-Dawley rats.

TMAO: A small molecule of great expectations.

Characterization of Disopyramide derivative ADD424042 as a non-cardiotoxic neuronal sodium channel blocker with broad-spectrum anticonvulsant activity in rodent seizure models.

Hypotensive effect of S-adenosyl-L-methionine in hypertensive rats is reduced by autonomic ganglia and KATP channel blockers.

Electrocardiography in rats: a comparison to human.

Intracolonic hydrogen sulfide lowers blood pressure in rats.

Repeated restraint stress produces acute and chronic changes in hemodynamic parameters in rats.

Gut Bacteria and Hydrogen Sulfide: The New Old Players in Circulatory System Homeostasis.

The gut-blood barrier permeability - A new marker in cardiovascular and metabolic diseases?

Renal denervation decreases blood pressure and renal tyrosine hydroxylase but does not augment the effect of hypotensive drugs.

Narcolepsy type 1 and hypersomnia associated with a psychiatric disorder show different slow wave activity dynamics.

Parenteral NaS, a fast-releasing HS donor, but not GYY4137, a slow-releasing HS donor, lowers blood pressure in rats.

NaS, a fast-releasing HS donor, given as suppository lowers blood pressure in rats.

Insomnia in Pregnancy Is Associated With Depressive Symptoms and Eating at Night.

The Upright Body Position Increases Translaminar Pressure Gradient in Normotensive and Hypertensive Rats.

Colonic hydrogen sulfide produces portal hypertension and systemic hypotension in rats.

Gut bacteria-derived molecules as mediators and markers in cardiovascular diseases. The role of the gut-blood barrier.

A common humoral background of intraocular and arterial blood pressure dysregulation.

Hypertension in rats is associated with an increased permeability of the colon to TMA, a gut bacteria metabolite.

Indole and indoxyl sulfate, gut bacteria metabolites of tryptophan, change arterial blood pressure via peripheral and central mechanisms in rats.

Trimethylamine N-oxide: A harmful, protective or diagnostic marker in lifestyle diseases?

Enalapril decreases rat plasma concentration of TMAO, a gut bacteria-derived cardiovascular marker.

High salt intake increases plasma trimethylamine N-oxide (TMAO) concentration and produces gut dysbiosis in rats.

Indoles - Gut Bacteria Metabolites of Tryptophan with Pharmacotherapeutic Potential.

Colonic indole, gut bacteria metabolite of tryptophan, increases portal blood pressure in rats.

Butyrate, a Gut Bacterial Metabolite, Lowers Intraocular Pressure in Normotensive But Not in Hypertensive Rats.

Gut Mycobiota and Fungal Metabolites in Human Homeostasis.

Salivary Hydrogen Sulfide Measured with a New Highly Sensitive Self-Immolative Coumarin-Based Fluorescent Probe.

CHRONIC, LOW-DOSE TMAO TREATMENT REDUCES DIASTOLIC DYSFUNCTION AND HEART FIBROSIS IN HYPERTENSIVE RATS.