Beau R. Webber

TALEN-based gene correction for epidermolysis bullosa.

DNA methylation of Runx1 regulatory regions correlates with transition from primitive to definitive hematopoietic potential in vitro and in vivo.

Trp53 haploinsufficiency modifies EGFR-driven peripheral nerve sheath tumorigenesis.

Fanconi anemia gene editing by the CRISPR/Cas9 system.

From marrow to matrix: novel gene and cell therapies for epidermolysis bullosa.

In Vitro T-Cell Generation From Adult, Embryonic, and Induced Pluripotent Stem Cells: Many Roads to One Destination.

Evaluation of TCR Gene Editing Achieved by TALENs, CRISPR/Cas9, and megaTAL Nucleases.

CRISPR/Cas9 Targeted Gene Editing and Cellular Engineering in Fanconi Anemia.

Angiotensin receptor blockade mediated amelioration of mucopolysaccharidosis type I cardiac and craniofacial pathology.

CRISPR/Cas9-based genetic correction for recessive dystrophic epidermolysis bullosa.

Aryl hydrocarbon receptor inhibition promotes hematolymphoid development from human pluripotent stem cells.

CRISPR/Cas9-Mediated Correction of the FANCD1 Gene in Primary Patient Cells.

Dermatopontin in Bone Marrow Extracellular Matrix Regulates Adherence but Is Dispensable for Murine Hematopoietic Cell Maintenance.

Rapid generation of Col7a1 mouse model of recessive dystrophic epidermolysis bullosa and partial rescue via immunosuppressive dermal mesenchymal stem cells.

Rapid DNA replication origin licensing protects stem cell pluripotency.

CRISPR/Cas9-Based Cellular Engineering for Targeted Gene Overexpression.

Engineering of Primary Human B cells with CRISPR/Cas9 Targeted Nuclease.

Highly multiplexed genome engineering using CRISPR/Cas9 gRNA arrays.

EditR: A Method to Quantify Base Editing from Sanger Sequencing.

Base Editor Correction of COL7A1 in Recessive Dystrophic Epidermolysis Bullosa Patient-Derived Fibroblasts and iPSCs.

A Genetically Engineered Primary Human Natural Killer Cell Platform for Cancer Immunotherapy.

Highly efficient multiplex human T cell engineering without double-strand breaks using Cas9 base editors.

Author Correction: Highly efficient multiplex human T cell engineering without double-strand breaks using Cas9 base editors.

Efficient targeted integration directed by short homology in zebrafish and mammalian cells.