Mi-Kyung Shin

Intermittent hypoxia inhibits clearance of triglyceride-rich lipoproteins and inactivates adipose lipoprotein lipase in a mouse model of sleep apnoea.

Effects of different acute hypoxic regimens on tissue oxygen profiles and metabolic outcomes.

Acute hypoxia induces hypertriglyceridemia by decreasing plasma triglyceride clearance in mice.

Metabolic consequences of high-fat diet are attenuated by suppression of HIF-1α.

Thermoneutrality modifies the impact of hypoxia on lipid metabolism.

Chronic intermittent hypoxia induces atherosclerosis via activation of adipose angiopoietin-like 4.

Effect of chronic intermittent hypoxia on triglyceride uptake in different tissues.

Carotid body denervation prevents fasting hyperglycemia during chronic intermittent hypoxia.

The effect of adrenal medullectomy on metabolic responses to chronic intermittent hypoxia.

Intermittent hypoxia-induced glucose intolerance is abolished by α-adrenergic blockade or adrenal medullectomy.

Lysyl Oxidase as a Serum Biomarker of Liver Fibrosis in Patients with Severe Obesity and Obstructive Sleep Apnea.

Adipose HIF-1α causes obesity by suppressing brown adipose tissue thermogenesis.

Hepatocyte Hypoxia Inducible Factor-1 Mediates the Development of Liver Fibrosis in a Mouse Model of Nonalcoholic Fatty Liver Disease.

Effect of adrenal medullectomy on metabolic responses to chronic intermittent hypoxia in the frequently sampled intravenous glucose tolerance test.

High fat diet induces airway hyperresponsiveness in mice.

Sleep-disordered breathing in C57BL/6J mice with diet-induced obesity.

Leptin acts in the carotid bodies to increase minute ventilation during wakefulness and sleep and augment the hypoxic ventilatory response.

Intranasal Leptin Relieves Sleep-disordered Breathing in Mice with Diet-induced Obesity.

Caloric restriction prevents the development of airway hyperresponsiveness in mice on a high fat diet.

Leptin Induces Hypertension Acting on Transient Receptor Potential Melastatin 7 Channel in the Carotid Body.