Jan Hendrik Niess

CX3CR1-mediated dendritic cell access to the intestinal lumen and bacterial clearance.

Lamina propria dendritic cells in the physiology and pathology of the gastrointestinal tract.

Dendritic cells in the recognition of intestinal microbiota.

Commensal gut flora drives the expansion of proinflammatory CD4 T cells in the colonic lamina propria under normal and inflammatory conditions.

Genotype-phenotype analysis of the CXCL16 p.Ala181Val polymorphism in inflammatory bowel disease.

Strongyloides stercoralis: a rare cause of obstructive duodenal stenosis.

Role of mucosal dendritic cells in inflammatory bowel disease.

Enteric flora expands gut lamina propria CX3CR1+ dendritic cells supporting inflammatory immune responses under normal and inflammatory conditions.

Selection of bifidobacteria based on adhesion and anti-inflammatory capacity in vitro for amelioration of murine colitis.

Safety of probiotic Escherichia coli strain Nissle 1917 depends on intestinal microbiota and adaptive immunity of the host.

Role of gut-resident dendritic cells in inflammatory bowel disease.

Expression and regulation of the chemokine CXCL16 in Crohn's disease and models of intestinal inflammation.

Gut microbiota, probiotics and inflammatory bowel disease.

What are CX3CR1+ mononuclear cells in the intestinal mucosa?

Identification of CCR9- murine plasmacytoid DC precursors with plasticity to differentiate into conventional DCs.

NOD2 polymorphism predicts response to treatment in Crohn's disease--first steps to a personalized therapy.

CD69 regulates type I IFN-induced tolerogenic signals to mucosal CD4 T cells that attenuate their colitogenic potential.

Tissue-specific differentiation of a circulating CCR9- pDC-like common dendritic cell precursor.

Metabolic alkalosis, acute renal failure and epileptic seizures as unusual manifestations of an upside-down stomach.

Anti-TNF and skin inflammation in IBD: a new paradox in gastroenterology?

Age-of-onset-dependent influence of NOD2 gene variants on disease behaviour and treatment in Crohn's disease.

The early activation marker CD69 regulates the expression of chemokines and CD4 T cell accumulation in intestine.

CD14-dependent monocyte isolation enhances phagocytosis of listeria monocytogenes by proinflammatory, GM-CSF-derived macrophages.

CD69 is the crucial regulator of intestinal inflammation: a new target molecule for IBD treatment?

A Pleiotropic Missense Variant in SLC39A8 Is Associated With Crohn's Disease and Human Gut Microbiome Composition.

The Stimulation of Macrophages with TLR Ligands Supports Increased IL-19 Expression in Inflammatory Bowel Disease Patients and in Colitis Models.