Mariko Omatsu-Kanbe

Inhibition of store-operated Ca2+ entry by extracellular ATP in rat brown adipocytes.

Rapidly and slowly activating components of delayed rectifier K(+) current in guinea-pig sino-atrial node pacemaker cells.

Multiple P2 receptors contribute to a transient increase in intracellular Ca2+ concentration in ATP-stimulated rat brown adipocytes.

Properties of the Na+/K+ pump current in small neurons from adult rat dorsal root ganglia.

Cytosolic Ca2+ under high glucose with suppressed Na+/K+ pump activity in rat sensory neurons.

Actin filaments play a permissive role in the inhibition of store-operated Ca2+ entry by extracellular ATP in rat brown adipocytes.

Effect of ATP on preadipocyte migration and adipocyte differentiation by activating P2Y receptors in 3T3-L1 cells.

In vitro expression of natriuretic peptides in cardiomyocytes differentiated from monkey embryonic stem cells.

Angiotensin II potentiates the slow component of delayed rectifier K+ current via the AT1 receptor in guinea pig atrial myocytes.

Monkey embryonic stem cells differentiate into adipocytes in vitro.

Effect of protein kinase Cbeta inhibitor on Ca2+ homeostasis in diabetic sensory neurons.

Inhibitory action of protein kinase Cbeta inhibitor on tetrodotoxin-resistant Na+ current in small dorsal root ganglion neurons in diabetic rats.

Swelling-activated Cl(-) current in isolated rabbit articular chondrocytes: inhibition by arachidonic Acid.

A novel type of self-beating cardiomyocytes in adult mouse ventricles.

Angiotensin II type 1 receptor mediates partially hyposmotic-induced increase of I (Ks) current in guinea pig atrium.

Self-beating atypically shaped cardiomyocytes survive a long-term postnatal development while preserving the expression of fetal cardiac genes in mice.

Characterization of the rapidly activating delayed rectifier potassium current, I (Kr), in HL-1 mouse atrial myocytes.

Ca2+ paradox injury mediated through TRPC channels in mouse ventricular myocytes.

Sevoflurane protects ventricular myocytes from Ca2+ paradox-mediated Ca2+ overload by blocking the activation of transient receptor potential canonical channels.

Autophagy is constitutively active in normal mouse sino-atrial nodal cells.

Inhibitory effects of sevoflurane on pacemaking activity of sinoatrial node cells in guinea-pig heart.

Postnatal developmental decline in IK1 in mouse ventricular myocytes isolated by the Langendorff perfusion method: comparison with the chunk method.

PKC-Mediated ZYG1 Phosphorylation Induces Fusion of Myoblasts as well as of Dictyostelium Cells.

Ischemic survival and constitutively active autophagy in self-beating atypically-shaped cardiomyocytes (ACMs): characterization of a new subpopulation of heart cells.

Sevoflurane protects ventricular myocytes against oxidative stress-induced cellular Ca2+ overload and hypercontracture.

Novel intracellular mediator of adiponectin secretion from adipocytes.

Prion protein- and cardiac troponin T-marked interstitial cells from the adult myocardium spontaneously develop into beating cardiomyocytes.

Phosphatidylinositol4-phosphate 5-kinase prevents the decrease in the HERG potassium current induced by Gq protein-coupled receptor stimulation.

Identification of cardiac progenitors that survive in the ischemic human heart after ventricular myocyte death.

Novel intracellular transport-refractory mutations in KCNH2 identified in patients with symptomatic long QT syndrome.

Postnatal developmental changes in the sensitivity of L-type Ca channel to inhibition by verapamil in a mouse heart model.

A simple antegrade perfusion method for isolating viable single cardiomyocytes from neonatal to aged mice.

Elevation of propofol sensitivity of cardiac I channel by KCNE1 polymorphism D85N.