Hiroshi Matsuura

Inhibition of store-operated Ca2+ entry by extracellular ATP in rat brown adipocytes.

Rapidly and slowly activating components of delayed rectifier K(+) current in guinea-pig sino-atrial node pacemaker cells.

Potentiation of slow component of delayed rectifier K(+) current by cGMP via two distinct mechanisms: inhibition of phosphodiesterase 3 and activation of protein kinase G.

Blocking action of chromanol 293B on the slow component of delayed rectifier K(+) current in guinea-pig sino-atrial node cells.

Multiple P2 receptors contribute to a transient increase in intracellular Ca2+ concentration in ATP-stimulated rat brown adipocytes.

Properties of the Na+/K+ pump current in small neurons from adult rat dorsal root ganglia.

Cytosolic Ca2+ under high glucose with suppressed Na+/K+ pump activity in rat sensory neurons.

Actin filaments play a permissive role in the inhibition of store-operated Ca2+ entry by extracellular ATP in rat brown adipocytes.

Functional role of hCngb3 in regulation of human cone cng channel: effect of rod monochromacy-associated mutations in hCNGB3 on channel function.

Regulation of cardiac IKs potassium current by membrane phosphatidylinositol 4,5-bisphosphate.

Responses of the sustained inward current to autonomic agonists in guinea-pig sino-atrial node pacemaker cells.

Regulation of the muscarinic K+ channel by extracellular ATP through membrane phosphatidylinositol 4,5-bisphosphate in guinea-pig atrial myocytes.

Open-state unblock characterizes acute inhibition of I potassium current by amiodarone in guinea pig ventricular myocytes.

Effect of ATP on preadipocyte migration and adipocyte differentiation by activating P2Y receptors in 3T3-L1 cells.

In vitro expression of natriuretic peptides in cardiomyocytes differentiated from monkey embryonic stem cells.

Angiotensin II potentiates the slow component of delayed rectifier K+ current via the AT1 receptor in guinea pig atrial myocytes.

Monkey embryonic stem cells differentiate into adipocytes in vitro.

Effect of protein kinase Cbeta inhibitor on Ca2+ homeostasis in diabetic sensory neurons.

Modulation of functional properties of KCNQ1 channel by association of KCNE1 and KCNE2.

Inhibitory action of protein kinase Cbeta inhibitor on tetrodotoxin-resistant Na+ current in small dorsal root ganglion neurons in diabetic rats.

Functional analysis of rod monochromacy-associated missense mutations in the CNGA3 subunit of the cone photoreceptor cGMP-gated channel.

Cinnamyl-3,4-dihydroxy-alpha-cyanocinnamate and nordihydroguaiaretic acid inhibit human Kv1.5 currents independently of lipoxygenase.

Hydroxyzine, a first generation H(1)-receptor antagonist, inhibits human ether-a-go-go-related gene (HERG) current and causes syncope in a patient with the HERG mutation.

Swelling-activated Cl(-) current in isolated rabbit articular chondrocytes: inhibition by arachidonic Acid.

A novel type of self-beating cardiomyocytes in adult mouse ventricles.

Angiotensin II type 1 receptor mediates partially hyposmotic-induced increase of I (Ks) current in guinea pig atrium.

Adrenergic regulation of the rapid component of delayed rectifier K+ current: implications for arrhythmogenesis in LQT2 patients.

Regulatory role of tyrosine phosphorylation in the swelling-activated chloride current in isolated rabbit articular chondrocytes.

Latent genetic backgrounds and molecular pathogenesis in drug-induced long-QT syndrome.

KCNE2 modulation of Kv4.3 current and its potential role in fatal rhythm disorders.

Self-beating atypically shaped cardiomyocytes survive a long-term postnatal development while preserving the expression of fetal cardiac genes in mice.

Characterization of the rapidly activating delayed rectifier potassium current, I (Kr), in HL-1 mouse atrial myocytes.

Ca2+ paradox injury mediated through TRPC channels in mouse ventricular myocytes.

Presence and functional role of the rapidly activating delayed rectifier K(+) current in left and right atria of adult mice.

Lysophosphatidylcholine enhances I(Ks) currents in cardiac myocytes through activation of G protein, PKC and Rho signaling pathways.

Atrioventricular block-induced Torsades de Pointes with clinical and molecular backgrounds similar to congenital long QT syndrome.

Sevoflurane protects ventricular myocytes from Ca2+ paradox-mediated Ca2+ overload by blocking the activation of transient receptor potential canonical channels.

Identification and functional characterization of KCNQ1 mutations around the exon 7-intron 7 junction affecting the splicing process.

Autophagy is constitutively active in normal mouse sino-atrial nodal cells.

17β-Oestradiol inhibits doxorubicin-induced apoptosis via block of the volume-sensitive Cl(-) current in rabbit articular chondrocytes.

Inhibitory effects of sevoflurane on pacemaking activity of sinoatrial node cells in guinea-pig heart.

GHGKHKNK octapeptide (P-5m) inhibits metastasis of HCCLM3 cell lines via regulation of MMP-2 expression in in vitro and in vivo studies.

Postnatal developmental decline in IK1 in mouse ventricular myocytes isolated by the Langendorff perfusion method: comparison with the chunk method.

Phenotype variability in patients carrying KCNJ2 mutations.

Ischemic survival and constitutively active autophagy in self-beating atypically-shaped cardiomyocytes (ACMs): characterization of a new subpopulation of heart cells.

Irbesartan-mediated AT1 receptor blockade attenuates hyposmotic-induced enhancement of I Ks current and prevents shortening of action potential duration in atrial myocytes.

Sevoflurane protects ventricular myocytes against oxidative stress-induced cellular Ca2+ overload and hypercontracture.

The COX-2 selective blocker etodolac inhibits TNFα-induced apoptosis in isolated rabbit articular chondrocytes.

A novel KCNQ1 missense mutation identified in a patient with juvenile-onset atrial fibrillation causes constitutively open IKs channels.

Remifentanil has a minimal direct effect on sinoatrial node pacemaker activity in the Guinea pig heart.

Cardiac channelopathies associated with infantile fatal ventricular arrhythmias: from the cradle to the bench.

A molecular mechanism for adrenergic-induced long QT syndrome.

Gain-of-function KCNH2 mutations in patients with Brugada syndrome.

Direct negative chronotropic action of desflurane on sinoatrial node pacemaker activity in the guinea pig heart.

Improved functional expression of human cardiac kv1.5 channels and trafficking-defective mutants by low temperature treatment.

Long QT syndrome type 8: novel CACNA1C mutations causing QT prolongation and variant phenotypes.

Ca2+/calmodulin potentiates I Ks in sinoatrial node cells by activating Ca2+/calmodulin-dependent protein kinase II.

Ionic mechanisms underlying the negative chronotropic action of propofol on sinoatrial node automaticity in guinea pig heart.

Novel intracellular mediator of adiponectin secretion from adipocytes.

Prion protein- and cardiac troponin T-marked interstitial cells from the adult myocardium spontaneously develop into beating cardiomyocytes.

Phosphatidylinositol4-phosphate 5-kinase prevents the decrease in the HERG potassium current induced by Gq protein-coupled receptor stimulation.

Interaction of propofol with voltage-gated human Kv1.5 channel through specific amino acids within the pore region.

Putative binding sites for arachidonic acid on the human cardiac Kv 1.5 channel.

Lidocaine induces ROCK-dependent membrane blebbing and subsequent cell death in rabbit articular chondrocytes.

Serum albumin attenuates the open-channel blocking effects of propofol on the human Kv1.5 channel.

Regulation of human cardiac Kv1.5 channels by extracellular acidification.

Contribution of a KCNH2 variant in genotyped long QT syndrome: Romano-Ward syndrome under double mutations and acquired long QT syndrome under heterozygote.

Identification of cardiac progenitors that survive in the ischemic human heart after ventricular myocyte death.

Ionic mechanisms of the action of anaesthetics on sinoatrial node automaticity.

Ca1.3 L-type Ca channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na current.

The organic anion transporter SLCO2A1 constitutes the core component of the Maxi-Cl channel.

Novel intracellular transport-refractory mutations in KCNH2 identified in patients with symptomatic long QT syndrome.

Heterogeneous functional expression of the sustained inward Na current in guinea pig sinoatrial node cells.

Interactions of Propofol With Human Voltage-gated Kv1.5 Channel Determined by Docking Simulation and Mutagenesis Analyses.

Transient Receptor Potential Canonical Channel Blockers Improve Ventricular Contractile Functions After Ischemia/Reperfusion in a Langendorff-perfused Mouse Heart Model.

A hERG mutation E1039X produced a synergistic lesion on I together with KCNQ1-R174C mutation in a LQTS family with three compound mutations.

Correction to: Heterogeneous functional expression of the sustained inward Na current in guinea pig sinoatrial node cells.

Postnatal developmental changes in the sensitivity of L-type Ca channel to inhibition by verapamil in a mouse heart model.

A simple antegrade perfusion method for isolating viable single cardiomyocytes from neonatal to aged mice.

P2X7 ionotropic receptor is functionally expressed in rabbit articular chondrocytes and mediates extracellular ATP cytotoxicity.

A novel mutation identified in a patient with Timothy syndrome without syndactyly exerts both marked loss- and gain-of-function effects.

Combined antitumor effects of P-5m octapeptide and 5-fluorouracil on a murine model of H22 hepatoma ascites.

Long-term 4-AP treatment facilitates functional expression of human Kv1.5 channel.

A trafficking-deficient KCNQ1 mutation, T587M, causes a severe phenotype of long QT syndrome by interfering with intracellular hERG transport.

Identification of Verapamil Binding Sites Within Human Kv1.5 Channel Using Mutagenesis and Docking Simulation.

A de novo gain-of-function KCND3 mutation in early repolarization syndrome.

Anesthetic Management of a Patient With Type 1 Long QT Syndrome Using Combined Epidural-Spinal Anesthesia for Caesarean Section: Perioperative Approach Based on Ion Channel Function.

Identification of transmembrane protein 168 mutation in familial Brugada syndrome.

Open-channel blocking action of volatile anaesthetics desflurane and sevoflurane on human voltage-gated K 1.5 channel.

A computational analysis of the effect of sevoflurane in a human ventricular cell model of long QT syndrome: Importance of repolarization reserve in the QT-prolonging effect of sevoflurane.

Positive Inotropic Effects of ATP Released the Maxi-Anion Channel in Langendorff-Perfused Mouse Hearts Subjected to Ischemia-Reperfusion.

Elevation of propofol sensitivity of cardiac I channel by KCNE1 polymorphism D85N.