Ling-Gang Wu

Single and multiple vesicle fusion induce different rates of endocytosis at a central synapse.

Capacitance measurements at the calyx of Held in the medial nucleus of the trapezoid body.

Isoflurane inhibits transmitter release and the presynaptic action potential.

Kinetic regulation of vesicle endocytosis at synapses.

The decrease in the presynaptic calcium current is a major cause of short-term depression at a calyx-type synapse.

Activity-dependent acceleration of endocytosis at a central synapse.

Two modes of fusion pore opening revealed by cell-attached recordings at a synapse.

The origin of quantal size variation: vesicular glutamate concentration plays a significant role.

Role of Ca(2+) channels in short-term synaptic plasticity.

Rapid bulk endocytosis and its kinetics of fission pore closure at a central synapse.

The debate on the kiss-and-run fusion at synapses.

Modes of vesicle retrieval at ribbon synapses, calyx-type synapses, and small central synapses.

GTP-independent rapid and slow endocytosis at a central synapse.

Compound vesicle fusion increases quantal size and potentiates synaptic transmission.

Small voltage changes at nerve terminals travel up axons to affect action potential initiation.

Ca(2+) and calmodulin initiate all forms of endocytosis during depolarization at a nerve terminal.

Location matters: synaptotagmin helps place vesicles near calcium channels.

Rapid endocytosis does not recycle vesicles within the readily releasable pool.

Endocytosis and clathrin-uncoating defects at synapses of auxilin knockout mice.

The role of calcium/calmodulin-activated calcineurin in rapid and slow endocytosis at central synapses.

Post-tetanic potentiation is caused by two signalling mechanisms affecting quantal size and quantal content.

A membrane pool retrieved via endocytosis overshoot at nerve terminals: a study of its retrieval mechanism and role.

Cysteine string protein α: a new role in vesicle recycling.

Calcium-channel number critically influences synaptic strength and plasticity at the active zone.

Conditional expression of Parkinson's disease-related mutant α-synuclein in the midbrain dopaminergic neurons causes progressive neurodegeneration and degradation of transcription factor nuclear receptor related 1.

Voltage-dependent calcium channels at the plasma membrane, but not vesicular channels, couple exocytosis to endocytosis.

SNARE proteins synaptobrevin, SNAP-25, and syntaxin are involved in rapid and slow endocytosis at synapses.

Most vesicles in a central nerve terminal participate in recycling.

The SNARE proteins SNAP25 and synaptobrevin are involved in endocytosis at hippocampal synapses.

Exocytosis and endocytosis: modes, functions, and coupling mechanisms.

Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis.

Presynaptic glycine receptors as a potential therapeutic target for hyperekplexia disease.

The yin and yang of calcium effects on synaptic vesicle endocytosis.

Post-fusion structural changes and their roles in exocytosis and endocytosis of dense-core vesicles.

Calcineurin is universally involved in vesicle endocytosis at neuronal and nonneuronal secretory cells.

Senescence impairs direct conversion of human somatic cells to neurons.

Calcineurin upregulates local Ca(2+) signaling through ryanodine receptor-1 in airway smooth muscle cells.

Conversion of fibroblasts to neural cells by p53 depletion.

Erratum: Post-fusion structural changes and their roles in exocytosis and endocytosis of dense-core vesicles.

Brain-derived neurotrophic factor inhibits calcium channel activation, exocytosis, and endocytosis at a central nerve terminal.

Fusion of lysosomes with secretory organelles leads to uncontrolled exocytosis in the lysosomal storage disease mucolipidosis type IV.

Suppression of agrin-22 production and synaptic dysfunction in Cln1 (-/-) mice.

The calyx of Held in the auditory system: Structure, function, and development.

α-Synuclein Mutation Inhibits Endocytosis at Mammalian Central Nerve Terminals.

Hemi-fused structure mediates and controls fusion and fission in live cells.

Actin dynamics provides membrane tension to merge fusing vesicles into the plasma membrane.

Actin Is Crucial for All Kinetically Distinguishable Forms of Endocytosis at Synapses.

Membrane Tension Inhibits Rapid and Slow Endocytosis in Secretory Cells.

Visualization of Membrane Pore in Live Cells Reveals a Dynamic-Pore Theory Governing Fusion and Endocytosis.

Reprogramming glioblastoma multiforme cells into neurons by protein kinase inhibitors.

New observations in neuroscience using superresolution microscopy.

Shisa7 is a GABA receptor auxiliary subunit controlling benzodiazepine actions.

Protein Kinase C and Calmodulin Serve As Calcium Sensors for Calcium-Stimulated Endocytosis at Synapses.

Vesicle Shrinking and Enlargement Play Opposing Roles in the Release of Exocytotic Contents.

Interleukin-33 Promotes Serotonin Release from Enterochromaffin Cells for Intestinal Homeostasis.

Presynaptic Kv3 channels are required for fast and slow endocytosis of synaptic vesicles.

Preformed Ω-profile closure and kiss-and-run mediate endocytosis and diverse endocytic modes in neuroendocrine chromaffin cells.

Retraction Note: Reprogramming glioblastoma multiforme cells into neurons by protein kinase inhibitors.

Clathrin-mediated endocytosis cooperates with bulk endocytosis to generate vesicles.

Dynamin 1 controls vesicle size and endocytosis at hippocampal synapses.