Padmamalini Baskaran
School of Pharmacy
University of Wyoming
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Desensitization of soluble guanylyl cyclase, the NO receptor, by S-nitrosylation.
Proceedings of the National Academy of Sciences of the United States of America Jul, 2007 | Pubmed ID: 17636120
PAS-mediated dimerization of soluble guanylyl cyclase revealed by signal transduction histidine kinase domain crystal structure.
The Journal of biological chemistry Jan, 2008 | Pubmed ID: 18006497
Structure of cinaciguat (BAY 58-2667) bound to Nostoc H-NOX domain reveals insights into heme-mimetic activation of the soluble guanylyl cyclase.
The Journal of biological chemistry Jul, 2010 | Pubmed ID: 20463019
Aspartate 102 in the heme domain of soluble guanylyl cyclase has a key role in NO activation.
Biochemistry May, 2011 | Pubmed ID: 21491881
Inhibition of nitric oxide-stimulated vasorelaxation by carbon monoxide-releasing molecules.
Arteriosclerosis, thrombosis, and vascular biology Nov, 2011 | Pubmed ID: 21836072
Identification of residues in the heme domain of soluble guanylyl cyclase that are important for basal and stimulated catalytic activity.
PloS one , 2011 | Pubmed ID: 22096512
Soluble guanylyl cyclase is a target of angiotensin II-induced nitrosative stress in a hypertensive rat model.
American journal of physiology. Heart and circulatory physiology Sep, 2012 | Pubmed ID: 22730391
Protein disulfide-isomerase interacts with soluble guanylyl cyclase via a redox-based mechanism and modulates its activity.
The Biochemical journal May, 2013 | Pubmed ID: 23477350
Effects of enzymatically inactive recombinant botulinum neurotoxin type A at the mouse neuromuscular junctions.
Toxicon : official journal of the International Society on Toxinology Sep, 2013 | Pubmed ID: 23810945
Cyclophilin A is required for angiotensin II-induced p47phox translocation to caveolae in vascular smooth muscle cells.
Arteriosclerosis, thrombosis, and vascular biology Sep, 2013 | Pubmed ID: 23846495
Acute and chronic effects of botulinum neurotoxin a on the mammalian neuromuscular junction.
Muscle & nerve Aug, 2014 | Pubmed ID: 24218344
Acetylation of cyclophilin A is required for its secretion and vascular cell activation.
Cardiovascular research Mar, 2014 | Pubmed ID: 24293519
Cyclophilin A is an important mediator of platelet function by regulating integrin αIIbβ3 bidirectional signalling.
Thrombosis and haemostasis May, 2014 | Pubmed ID: 24429998
Capsaicin modulates acetylcholine release at the myoneural junction.
European journal of pharmacology Dec, 2014 | Pubmed ID: 25446918
The TRPM8 protein is a testosterone receptor: I. Biochemical evidence for direct TRPM8-testosterone interactions.
The Journal of biological chemistry Jan, 2015 | Pubmed ID: 25480783
The TRPM8 protein is a testosterone receptor: II. Functional evidence for an ionotropic effect of testosterone on TRPM8.
The Journal of biological chemistry Jan, 2015 | Pubmed ID: 25480785
Capsaicin induces browning of white adipose tissue and counters obesity by activating TRPV1 channel-dependent mechanisms.
British journal of pharmacology Aug, 2016 | Pubmed ID: 27174467
Preparation and Evaluation of PLGA-Coated Capsaicin Magnetic Nanoparticles.
Pharmaceutical research Jun, 2017 | Pubmed ID: 28326459
Perturbation to Cholesterol at the Neuromuscular Junction confers botulinum neurotoxin A sensitivity to neonatal mice.
Toxicological sciences : an official journal of the Society of Toxicology Jun, 2017 | Pubmed ID: 28645175
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