Department of Child Health
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The p38α MAPK regulates microglial responsiveness to diffuse traumatic brain injury.
The Journal of neuroscience : the official journal of the Society for Neuroscience Apr, 2013 | Pubmed ID: 23554495
Using anesthetics and analgesics in experimental traumatic brain injury.
Lab animal Aug, 2013 | Pubmed ID: 23877609
Diffuse brain injury induces acute post-traumatic sleep.
PloS one , 2014 | Pubmed ID: 24416145
Recovery of neurological function despite immediate sleep disruption following diffuse brain injury in the mouse: clinical relevance to medically untreated concussion.
Sleep , 2014 | Pubmed ID: 24899763
Resolvins AT-D1 and E1 differentially impact functional outcome, post-traumatic sleep, and microglial activation following diffuse brain injury in the mouse.
Brain, behavior, and immunity Jan, 2015 | Pubmed ID: 25585137
Experimental diffuse brain injury results in regional alteration of gross vascular morphology independent of neuropathology.
Brain injury , 2016 | Pubmed ID: 26646974
Aging with a traumatic brain injury: Could behavioral morbidities and endocrine symptoms be influenced by microglial priming?
Brain, behavior, and immunity Jan, 2017 | Pubmed ID: 26975888
Lipid mediators of inflammation in neurological injury: shifting the balance toward resolution.
Neural regeneration research Jan, 2016 | Pubmed ID: 26981087
Diffuse traumatic brain injury induces prolonged immune dysregulation and potentiates hyperalgesia following a peripheral immune challenge.
Molecular pain , 2016 | Pubmed ID: 27178244
Midline (Central) Fluid Percussion Model of Traumatic Brain Injury.
Methods in molecular biology (Clifton, N.J.) , 2016 | Pubmed ID: 27604721
Partial cage division significantly reduces aggressive behavior in male laboratory mice.
Laboratory animals Aug, 2018 | Pubmed ID: 29409371
Midline (central) fluid percussion model of traumatic brain injury in pediatric and adolescent rats.
Journal of neurosurgery. Pediatrics Jul, 2018 | Pubmed ID: 29676680
Novel TNF receptor-1 inhibitors identified as potential therapeutic candidates for traumatic brain injury.
Journal of neuroinflammation May, 2018 | Pubmed ID: 29789012
Tabitha R.F. Green1,2,3,
J. Bryce Ortiz1,
Jordan L. Harrison5,
Jonathan Lifshitz1,3,4,
Rachel K. Rowe1,3,4
1Department of Child Health, University of Arizona College of Medicine,
2Department of Biological Sciences, University of Bath,
3, BARROW Neurological Institute at Phoenix Children's Hospital,
4, Phoenix Veteran Affairs Healthcare System,
5Department of Basic Medical Sciences, University of Arizona College of Medicine
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