Panchanan Maiti

Hypobaric hypoxia induces oxidative stress in rat brain.

Hypobaric hypoxia damages the hippocampal pyramidal neurons in the rat brain.

Hypobaric hypoxia induces dendritic plasticity in cortical and hippocampal pyramidal neurons in rat brain.

High altitude memory impairment is due to neuronal apoptosis in hippocampus, cortex and striatum.

Nitric oxide system is involved in hypobaric hypoxia-induced oxidative stress in rat brain.

Acetylcholinesterase inhibitors enhance cognitive functions in rats following hypobaric hypoxia.

Despite its role in assembly, methionine 35 is not necessary for amyloid beta-protein toxicity.

Cholinesterase inhibitors ameliorate spatial learning deficits in rats following hypobaric hypoxia.

Surprising toxicity and assembly behaviour of amyloid β-protein oxidized to sulfone.

Possible role of cholinesterase inhibitors on memory consolidation following hypobaric hypoxia of rats.

Role of cholinergic markers on memory function of rats exposed to hypobaric hypoxia.

Induction of methionine-sulfoxide reductases protects neurons from amyloid β-protein insults in vitro and in vivo.

Role of Epigenetics in Stem Cell Proliferation and Differentiation: Implications for Treating Neurodegenerative Diseases.

A comparative study of dietary curcumin, nanocurcumin, and other classical amyloid-binding dyes for labeling and imaging of amyloid plaques in brain tissue of 5×-familial Alzheimer's disease mice.

CRISPR-Cas9 Mediated Gene-Silencing of the Mutant Huntingtin Gene in an In Vitro Model of Huntington's Disease.

Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer's disease.

Use of Curcumin, a Natural Polyphenol for Targeting Molecular Pathways in Treating Age-Related Neurodegenerative Diseases.