Lilin Ye

The transcription factor TCF-1 initiates the differentiation of T(FH) cells during acute viral infection.

Follicular CXCR5- expressing CD8(+) T cells curtail chronic viral infection.

ABHD5 interacts with BECN1 to regulate autophagy and tumorigenesis of colon cancer independent of PNPLA2.

mTOR Promotes Antiviral Humoral Immunity by Differentially Regulating CD4 Helper T Cell and B Cell Responses.

Efficient control of chronic LCMV infection by a CD4 T cell epitope-based heterologous prime-boost vaccination in a murine model.

Antigen-specific CD8 T cell feedback activates NLRP3 inflammasome in antigen-presenting cells through perforin.

Mammalian target of rapamycin complex 1 signalling is essential for germinal centre reaction.

Oral administration of visceral adipose tissue antigens ameliorates metabolic disorders in mice and elevates visceral adipose tissue-resident CD4CD25Foxp3 regulatory T cells.

The differential organogenesis and functionality of two liver-draining lymph nodes in mice.

The Kinase mTORC1 Promotes the Generation and Suppressive Function of Follicular Regulatory T Cells.

Dichotomous Roles of Programmed Cell Death 1 on HIV-Specific CXCR5 and CXCR5 CD8 T Cells during Chronic HIV Infection.

Analyzing Mouse B Cell Responses Specific to LCMV Infection.

Flow Cytometry Analysis of mTOR Signaling in Antigen-Specific B Cells.

Patient-shared TCRβ-CDR3 clonotypes correlate with favorable prognosis in chronic hepatitis B.

Differentiation and Function of Follicular CD8 T Cells During Human Immunodeficiency Virus Infection.

The Kinase Complex mTOR Complex 2 Promotes the Follicular Migration and Functional Maturation of Differentiated Follicular Helper CD4 T Cells During Viral Infection.

B7S1, a novel candidate for anti-tumor checkpoint blockade immunotherapy.

Late-stage tumors induce anemia and immunosuppressive extramedullary erythroid progenitor cells.

A Portrait of CXCR5 Follicular Cytotoxic CD8 T cells.

Crosstalks between mTORC1 and mTORC2 variagate cytokine signaling to control NK maturation and effector function.

Liver-Resident NK Cells Control Antiviral Activity of Hepatic T Cells via the PD-1-PD-L1 Axis.

Genome-wide analysis identifies NR4A1 as a key mediator of T cell dysfunction.

The Transcription Factor TCF1 Preserves the Effector Function of Exhausted CD8 T Cells During Chronic Viral Infection.

Molecular Basis of the Differentiation and Function of Virus Specific Follicular Helper CD4 T Cells.

The histone methyltransferase EZH2 primes the early differentiation of follicular helper T cells during acute viral infection.

Ceria nanoparticles promoted the cytotoxic activity of CD8 T cells by activating NF-κB signaling.

The Transcription Factor T-Bet Is Required for Optimal Type I Follicular Helper T Cell Maintenance During Acute Viral Infection.

Cutting Edge: Transcription Factor BCL6 Is Required for the Generation, but Not Maintenance, of Memory CD8 T Cells in Acute Viral Infection.

Sclerostin domain-containing protein 1 is dispensable for the differentiation of follicular helper and follicular regulatory T cells during acute viral infection.

ZIKV infection induces robust Th1-like Tfh cell and long-term protective antibody responses in immunocompetent mice.

DAPK1 (death associated protein kinase 1) mediates mTORC1 activation and antiviral activities in CD8 T cells.

Improving the immunogenicity and protective efficacy of a whole-killed malaria blood-stage vaccine by chloroquine.

T cell immune response within B-cell follicles.

Human monoclonal antibodies block the binding of SARS-CoV-2 spike protein to angiotensin converting enzyme 2 receptor.

CD49aCD49b NK cells induced by viral infection reflect an activated state of conventional NK cells.

Bcl6 Preserves the Suppressive Function of Regulatory T Cells During Tumorigenesis.

Disease severity dictates SARS-CoV-2-specific neutralizing antibody responses in COVID-19.

The Epigenetic Regulator EZH2 Instructs CD4 T Cell Response to Acute Viral Infection via Coupling of Cell Expansion and Metabolic Fitness.

METTL3-dependent mA modification programs T follicular helper cell differentiation.

The dichotomous and incomplete adaptive immunity in COVID-19 patients with different disease severity.

The lncRNA Snhg1-Vps13D vesicle trafficking system promotes memory CD8 T cell establishment via regulating the dual effects of IL-7 signaling.

The metabolic hormone leptin promotes the function of T cells and supports vaccine responses.

Sensitivity of SARS-CoV-2 Variants to Neutralization by Convalescent Sera and a VH3-30 Monoclonal Antibody.

A novel linear and broadly neutralizing peptide in the SARS-CoV-2 S2 protein for universal vaccine development.

The kinase complex mTORC2 promotes the longevity of virus-specific memory CD4 T cells by preventing ferroptosis.

Differential expression of inhibitory receptor NKG2A distinguishes disease-specific exhausted CD8 T cells.

Prompt Antiviral Action of Pulmonary CD8+ T Cells Is Mediated by Rapid IFN-γ Induction and Its Downstream ISGs in the Lung.

A potent human monoclonal antibody with pan-neutralizing activities directly dislocates S trimer of SARS-CoV-2 through binding both up and down forms of RBD.

A novel strategy to investigate the factors regulating the Treg to Tfr transition during acute viral infection.

35B5 antibody potently neutralizes SARS-CoV-2 Omicron by disrupting the N-glycan switch via a conserved spike epitope.

CD4 T-cell epitope-based heterologous prime-boost vaccination potentiates anti-tumor immunity and PD-1/PD-L1 immunotherapy.

Nasal Spray of Neutralizing Monoclonal Antibody 35B5 Confers Potential Prophylaxis Against Severe Acute Respiratory Syndrome Coronavirus 2 Variants of Concern: A Small-Scale Clinical Trial.

Tumor-Specific CD4 T Cells Restrain Established Metastatic Melanoma by Developing Into Cytotoxic CD4 T Cells.

The Differentiation and Maintenance of SARS-CoV-2-Specific Follicular Helper T Cells.

A Bispecific Antibody Targeting RBD and S2 Potently Neutralizes SARS-CoV-2 Omicron and Other Variants of Concern.

EZH2 restricts DNA methylation and promotes T differentiation during acute viral infection.

The primordial differentiation of tumor-specific memory CD8 T cells as bona fide responders to PD-1/PD-L1 blockade in draining lymph nodes.

The m1A modification of tRNAs: a translational accelerator of T-cell activation.

Role of CXCR5 CD8 T cells in human immunodeficiency virus-1 infection.

The land-scape of immune response to monkeypox virus.

Bound to be perfect: Lck and T cell co-receptors.

Dopamine inhibits group 2 innate lymphoid cell-driven allergic lung inflammation by dampening mitochondrial activity.

Mechanisms of antigen-induced reversal of CNS inflammation in experimental demyelinating disease.

Tumor-specific memory CD8 T cells are strictly resident in draining lymph nodes during tumorigenesis.

A novel mAb broadly neutralizes SARS-CoV-2 VOCs in vitro and in vivo, including the Omicron variants.

Identification and Comparison of the Sialic Acid-Binding Domain Characteristics of Avian Coronavirus Infectious Bronchitis Virus Spike Protein.

The G allele of SNP rs3922 reduces the binding affinity between IGF2BP3 and CXCR5 correlating with a lower antibody production.

Author Correction: Tumor-specific memory CD8 T cells are strictly resident in draining lymph nodes during tumorigenesis.

PD-1/PD-L1 blockade restores tumor-induced COVID-19 vaccine bluntness.

An RBD bispecific antibody effectively neutralizes a SARS-CoV-2 Omicron variant.

The TRIM37 variants in Mulibrey nanism patients paralyze follicular helper T cell differentiation.

Aged hematopoietic stem cells entrap regulatory T cells to create a prosurvival microenvironment.

Dopamine Receptor 1 Impedes ILC2-Mediated Antitumor Immunity.

The role of CD4 T cells in tumor and chronic viral immune responses.

Functional diversification and dynamics of CAR-T cells in patients with B-ALL.

Role of tumor cell pyroptosis in anti-tumor immunotherapy.

Bile acids modified by the intestinal microbiota promote colorectal cancer growth by suppressing CD8 T cell effector functions.

An oncolytic virus delivering tumor-irrelevant bystander T cell epitopes induces anti-tumor immunity and potentiates cancer immunotherapy.