J. Marie Hardwick

Epstein-Barr virus BALF1 is a BCL-2-like antagonist of the herpesvirus antiapoptotic BCL-2 proteins.

Inhibitor specificity of recombinant and endogenous caspase-9.

BCL-2 and BAX protect adult mice from lethal Sindbis virus infection but do not protect spinal cord motor neurons or prevent paralysis.

Bax-type apoptotic proteins porate pure lipid bilayers through a mechanism sensitive to intrinsic monolayer curvature.

Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria.

BAK alters neuronal excitability and can switch from anti- to pro-death function during postnatal development.

Enhancing DNA vaccine potency by coadministration of DNA encoding antiapoptotic proteins.

Enhancing DNA vaccine potency by combining a strategy to prolong dendritic cell life with intracellular targeting strategies.

Modulation of synaptic transmission by the BCL-2 family protein BCL-xL.

Inhibition of translation and induction of apoptosis by Bunyaviral nonstructural proteins bearing sequence similarity to reaper.

A comparison of the properties of a Bcl-xL variant to the wild-type anti-apoptosis inhibitor in mammalian cell cultures.

Viral modulators of cell death provide new links to old pathways.

Regulation of cell death in the lymphoid system by Bcl-2 family proteins.

Cytosolic domain of the human mitochondrial fission protein fis1 adopts a TPR fold.

Aven and Bcl-xL enhance protection against apoptosis for mammalian cells exposed to various culture conditions.

Viral Bcl-2 homologs and their role in virus replication and associated diseases.

Neuronal apoptosis pathways in Sindbis virus encephalitis.

BAD is a pro-survival factor prior to activation of its pro-apoptotic function.

Proapoptotic N-truncated BCL-xL protein activates endogenous mitochondrial channels in living synaptic terminals.

Mitochondrial fission proteins regulate programmed cell death in yeast.

Mitochondrial programmed cell death pathways in yeast.

Exposure to hypoxia rapidly induces mitochondrial channel activity within a living synapse.

Calpain I induces cleavage and release of apoptosis-inducing factor from isolated mitochondria.

Modification of professional antigen-presenting cells with small interfering RNA in vivo to enhance cancer vaccine potency.

Magnetic resonance diffusion tensor microimaging reveals a role for Bcl-x in brain development and homeostasis.

Viruses activate a genetically conserved cell death pathway in a unicellular organism.

Actions of BAX on mitochondrial channel activity and on synaptic transmission.

Combining caspase and mitochondrial dysfunction inhibitors of apoptosis to limit cell death in mammalian cell cultures.

Zinc-dependent multi-conductance channel activity in mitochondria isolated from ischemic brain.

Enhanced cell culture performance using inducible anti-apoptotic genes E1B-19K and Aven in the production of a monoclonal antibody with Chinese hamster ovary cells.

Upgrading the BCL-2 network.

A Bcl-xL timer sets platelet life span.

A quorum on bacterial programmed cell death.

Bcl-xL inhibitor ABT-737 reveals a dual role for Bcl-xL in synaptic transmission.

Anti-apoptotic Bcl-2 Family Proteins Disassemble Ceramide Channels.

Bcl-xL induces Drp1-dependent synapse formation in cultured hippocampal neurons.

Mitochondrial death pathways in yeast and mammalian cells.

Unravelling the bcl-2 apoptosis code with a simple model system.

Aven-dependent activation of ATM following DNA damage.

Bcl-2 turns deadly.

Bcl-x L increases mitochondrial fission, fusion, and biomass in neurons.

Reliable method for detection of programmed cell death in yeast.

SnapShot: BCL-2 proteins.

The apoptosome at high resolution.

Seizure tests distinguish intermittent fasting from the ketogenic diet.

Mitochondrial involvement in cell death of non-mammalian eukaryotes.

Flying to a halt: Drosophila Aven arrests the cell cycle.

Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.

Bcl-xL regulates mitochondrial energetics by stabilizing the inner membrane potential.

N-terminally cleaved Bcl-xL mediates ischemia-induced neuronal death.

Multipolar functions of BCL-2 proteins link energetics to apoptosis.

The mTOR inhibitor rapamycin has limited acute anticonvulsant effects in mice.

A new view of the lethal apoptotic pore.

Evolution of Bcl-2 homology motifs: homology versus homoplasy.

The N-terminal helix of Bcl-xL targets mitochondria.

Unlatched BAX pairs for death.

Multiple functions of BCL-2 family proteins.

Quantification of genetically controlled cell death in budding yeast.

Genome-wide consequences of deleting any single gene.

Cell death in genome evolution.

In vivo CaspaseTracker biosensor system for detecting anastasis and non-apoptotic caspase activity.

Potent anti-seizure effects of D-leucine.

Redefining the BH3 Death Domain as a 'Short Linear Motif'.