Milena Z. Adzemovic

Fine-mapping resolves Eae23 into two QTLs and implicates ZEB1 as a candidate gene regulating experimental neuroinflammation in rat.

IL-22RA2 associates with multiple sclerosis and macrophage effector mechanisms in experimental neuroinflammation.

Expression of Ccl11 associates with immune response modulation and protection against neuroinflammation in rats.

Imatinib ameliorates neuroinflammation in a rat model of multiple sclerosis by enhancing blood-brain barrier integrity and by modulating the peripheral immune response.

Efficacy of vitamin D in treating multiple sclerosis-like neuroinflammation depends on developmental stage.

Acute treatment with valproic acid and l-thyroxine ameliorates clinical signs of experimental autoimmune encephalomyelitis and prevents brain pathology in DA rats.

Role of Tumor Pericytes in the Recruitment of Myeloid-Derived Suppressor Cells.

Presymptomatic activation of the PDGF-CC pathway accelerates onset of ALS neurodegeneration.

Expression of vascular endothelial growth factor (VEGF)-B and its receptor (VEGFR1) in murine heart, lung and kidney.

Lipocalin-2 as an Infection-Related Biomarker to Predict Clinical Outcome in Ischemic Stroke.

Functional genomics analysis of vitamin D effects on CD4+ T cells in vivo in experimental autoimmune encephalomyelitis ‬.

Widespread cortical demyelination of both hemispheres can be induced by injection of pro-inflammatory cytokines via an implanted catheter in the cortex of MOG-immunized rats.

Preclinical toxicological assessment of a novel monoclonal antibody targeting human platelet-derived growth factor CC (PDGF-CC) in PDGF-CChum mice.

Neuronal methylome reveals CREB-associated neuro-axonal impairment in multiple sclerosis.

C-type lectin receptors Mcl and Mincle control development of multiple sclerosis-like neuroinflammation.

Microglial autophagy-associated phagocytosis is essential for recovery from neuroinflammation.

Effect of Vitamin D on Experimental Autoimmune Neuroinflammation Is Dependent on Haplotypes Comprising Naturally Occurring Allelic Variants of CIITA ().

Blocking PDGF-CC signaling ameliorates multiple sclerosis-like neuroinflammation by inhibiting disruption of the blood-brain barrier.

Anti-CD20 treatment effectively attenuates cortical pathology in a rat model of widespread cortical demyelination.