Gregg A. Hadley

CD103 expression is required for destruction of pancreatic islet allografts by CD8(+) T cells.

Regulation of CD103 expression by CD8+ T cells responding to renal allografts.

Long-term islet graft survival in NOD mice by abrogation of recurrent autoimmunity.

TGF-{beta}-dependent CD103 expression by CD8(+) T cells promotes selective destruction of the host intestinal epithelium during graft-versus-host disease.

Critical role for CD103+CD8+ effectors in promoting tubular injury following allogeneic renal transplantation.

Abrogation of recurrent autoimmunity in the NOD mouse: A critical role for host interleukin 4.

Comparison of the portal vein and kidney subcapsule as sites for primate islet autotransplantation.

Expression of the decay-accelerating factor (CD55) in renal transplants--a possible prediction marker of allograft survival.

Induction of CD4(+)CD25(+) T regulatory cells with CD103 depletion.

Environmental and antigen receptor-derived signals support sustained surveillance of the lungs by pathogen-specific cytotoxic T lymphocytes.

CD103 deficiency prevents graft-versus-host disease but spares graft-versus-tumor effects mediated by alloreactive CD8 T cells.

Application of anti-CD103 immunotoxin for saving islet allograft in context of transplantation.

Regulation of acute graft-versus-host disease by microRNA-155.