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Merck Research Laboratory
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MyD88 primes macrophages for full-scale activation by interferon-gamma yet mediates few responses to Mycobacterium tuberculosis.
The Journal of experimental medicine Oct, 2003 | Pubmed ID: 14517275
Mycobacterium tuberculosis appears to lack alpha-ketoglutarate dehydrogenase and encodes pyruvate dehydrogenase in widely separated genes.
Molecular microbiology Aug, 2005 | Pubmed ID: 16045627
Expression of many immunologically important genes in Mycobacterium tuberculosis-infected macrophages is independent of both TLR2 and TLR4 but dependent on IFN-alphabeta receptor and STAT1.
Journal of immunology (Baltimore, Md. : 1950) Sep, 2005 | Pubmed ID: 16116224
Induction of macrophage-derived SLPI by Mycobacterium tuberculosis depends on TLR2 but not MyD88.
Immunology Nov, 2005 | Pubmed ID: 16236128
Dihydrolipoamide acyltransferase is critical for Mycobacterium tuberculosis pathogenesis.
Infection and immunity Jan, 2006 | Pubmed ID: 16368957
mRNA stability and antibody production in CHO cells: improvement through gene optimization.
Biotechnology journal Apr, 2010 | Pubmed ID: 20222103
Virulence of Mycobacterium tuberculosis depends on lipoamide dehydrogenase, a member of three multienzyme complexes.
Cell host & microbe Jan, 2011 | Pubmed ID: 21238944
Merck & Co., Inc
Shuangping Shi1,
Russ G.G. Condon1,
Liang Deng1,
Jason Saunders1,
Finn Hung1,
Yung-Shyeng Tsao1,
Zhong Liu1
1Merck Research Laboratory, Merck & Co., Inc
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